C(=O)O[C@H]1C[C@H](O)C=C2C=C[C@H](C)[C@H](CC[C@@H](O)C[C@@H](O)CC(O)=O)[C@H]21](https://cdn.shopify.com/s/files/1/0389/1897/9723/products/M10181_1621561464.gif?v=1742883139)
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Smiles: CC[C@H](C)C(=O)O[C@H]1C[C@H](O)C=C2C=C[C@H](C)[C@H](CC[C@@H](O)C[C@@H](O)CC(O)=O)[C@H]21
HPGDS inhibitor 1 has equal potency against purified HPGDS from human
EPZ005687 has greater than 500-fold selectivity against 15 other protein methyltransferases and has 50-fold selectivity against the closely related enzyme EZH1
1-5H3/t18-
CEP-33779 - CAS# 1257704-57-6 nshinone I Smiles: CC[C@H](C)C(=O)O[C@H]1C[C@H](O)C=C2C=C[C@H](C)[C@H](CC[C@@H](O)C[C@@H](O)CC(O)=O)[C@H]21CEP 33779 is a highly selective, orally active, small molecule inhibitor of JAK2. CEP 33779 induced regression of established colorectal tumors, reduced angiogenesis, and reduced proliferation of tumor cells. Tumor regression correlated with inhibition of STAT3 and NF B (RelA p65) activation in a CEP 33779 dose dependent manner. The ability of CEP 33779 to suppress growth of colorectal tumors by inhibiting the IL 6 JAK2 STAT3 signaling suggests a
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